Missed cues on anthropogenic causes of human disease, new insights into climate change and suppression of objective science

Some recent posts on the AgeofAutism.com website concerning autism and autism spectrum disorder (ASD) and perhaps chemical causation intrigued me enough to do some additional reading.

Specifically, the link  AofA polio/DDT published in 2011 caused me to embark on a rather circuitous path that eventually led to investigation of human health developments since the 1970/1980’s time frame.  There is a curious apparent synergy that also coincides with a former Mons@nto researcher quickly being placed in an editorial position at Elsevier after the Seralini paper (2012) attracted considerable attention, negative press and eventual retraction. It has curious and unnerving similarities to the processes which led to the Wakefield 1998 Lancet paper retraction and continued lawsuits,

My Reading Sequence:

1. Janine Roberts (initial polio/DDT connections, 2004)
2. Jim West (pre Roberts version of polio causality, 2003)
2A MS Biskind and Scobey papers (1949-1953)
2B Rachel Carson (Silent Spring, 1962)

3. Blaxill/Olmsted (AofA polio/DDT connection, 2011
4. BlaxillOlmsted (CA polio cases, 2014 -1)
5. Blaxill/Olmsted (CA polio cases 2014 -2)
6. Blaxill/Olmsted (CA polio cases 2014-3, link unreachable- to be updated)

7. Temekes (2012-2013 neonicotinoid pesticide PPT/information)
8. Seralini (2012, retracted paper linking glyphosate w/ mammalian health effects)
8A. Seralini 2012-13 (2014 comments to reviewers, answers to questions)

9.   Samsel and Seneff (2013 glyphosate and human health Entropy paper)
10. Seneff (W A Price Conference Feb 2014 powerpoint)

11. Mason 2014 data on glyphosate/worldwide human health

Each one of these links represents an interesting body of collective knowledge which has deep implications about human health degradation, economics,  and global ecosystem dysfunctioning, including climate change.

Suggestion: Seneff powerpt (10), Samsel & Seneff (9), Mason (11), and then go from there.
Dietary revisions as needed: go organic, gluten free, perhaps medication revisions, sunlight, exercise regimen, limited vaccination(s).


New take on obesity

An interesting read / take on obesity.  The author, Berreby, cites some studies that provide some greater context about how obesity is likely more than genetics of human origin, has some genesis in the everyday chemicals/compounds in our food stream, and the incessant scream of western capitalistic media & 24hr food availability.

The Obesity Era
As the American people got fatter, so did marmosets, vervet monkeys and mice. The problem may be bigger than any of us’…

Missing are maybe some key insights about climate change (earlier spring & later winters, competitor dieoff) [see Flannery’s take on climate change] contributing to bigger animals and perhaps the entry to our food stream of things like high fructose corn syrup which corresponds nicely to a start of the current obesity epidemic. ALso, the fact that a virus (AD-36, also reported as ADV-36 in most peer reviewed literature) may be a cofactor. Maybe, this also suggests that a vaccine (cool- just what we all need, more vaccinations) has been in development (search “ADV-36” “virus””patents”). This virus- more active when, or contributing to, immune inflammatory responses (Na & Nam http://jid.oxfordjournals.org/content/205/6/914.full), appears associated with respiratory infections & targets various tissues associated with adipose (fat) cells.

Key Pts
1. A report by the consulting firm McKinsey & Co predicted in May 2012 that ‘health and wellness’ would soon become a trillion-dollar global industry.

2….it’s obvious who is to blame for this frightening global blanket of lipids: it’s us, choosing over and over again, billions of times a day, to eat too much and exercise too little.

3.The personal choice theory & big government’s response: Michael Bloomberg, recently put it, defending his proposed ban on large cups for sugary drinks: ‘If you want to lose weight, don’t eat. This is not medicine, it’s thermodynamics. If you take in more than you use, you store it.’

4. The alternative ‘no personal responsibility’ theory: Yet the scientists who study the biochemistry of fat and the epidemiologists who track weight trends are not nearly as unanimous as Bloomberg makes out. In fact, many researchers believe that personal gluttony and laziness cannot be the entire explanation for humanity’s global weight gain.

‘The previous belief of many lay people and health professionals that obesity is simply
     the result of a lack of willpower and an inability to discipline eating habits is no longer 
     defensible.’’ Berraby

‘Virtually in every population of animals we looked at, that met our criteria, there
was the 
same upward trend,’ David Allison, biostatistician at U Alabama, Birmingham

5. Yet a number of researchers have come to believe, as Wells himself wrote earlier this year in the European Journal of Clinical Nutrition, that ‘all calories are not equal’. The problem with diets that are heavy in meat, fat or sugar is not solely that they pack a lot of calories into food; it is that they alter the biochemistry of fat storage and fat expenditure, tilting the body’s system in favour of fat storage. Wells notes, for example, that sugar, trans-fats and alcohol have all been linked to changes in ‘insulin signalling’, which affects how the body processes carbohydrates. This might sound like a merely technical distinction. In fact, it’s a paradigm shift: if the problem isn’t the number of calories but rather biochemical influences on the body’s fat-making and fat-storage processes, then sheer quantity of food or drink are not the all-controlling determinants of weight gain. If candy’s chemistry tilts you toward fat, then the fact that you eat it at all may be as important as the amount of it you consume.

We are, of course, surrounded by industrial chemicals. According to Frederick vom Saal, professor of biological sciences at the University of Missouri, an organic compound called bisphenol-A (or BPA) that is used in many household plastics has the property of altering fat regulation in lab animals.

Vaccines didn’t & probably won’t save us – look at the data

Vaccines Did Not Save Us – 2 Centuries Of Official Statistics
Source:  http://childhealthsafety.wordpress.com/graphs/
     An interesting perspective on the incidence of childhood disease over the last 200 years for UK, US & Australia.  Diseases were well on the decline due to better public health practices, water treatment, and wealth increases in these countries.  Measles and mumps vaccinations, in particular, had been identified  as unnecessary by UK in late 1970’s- at least it was being questioned.
download pdf

Where is the science showing potential dangers from excess vaccinations &
what would be the likely immune/neurological responses?
Blaylock 2008 has an interesting article & viewpoint with 105 references.

US Gov’t has been quietly settling many vaccine-caused neurological damage cases
What would be interesting to know is why aren’t the companies that make these vaccines being held liable.

 Andrew Wakefield’s Responses Published -from childhealthsafety:
“rubella virus is one of the few known causes of autism.” US Center for Disease Control.
“FAQs (frequently asked questions) about MMR Vaccine & Autism”  [ED 8/Apr/12: This is the web archive of the CDC page – you will need to search in or scroll down the page to see the text.  As papers cited on the original page by the CDC as evidence for no link with the vaccine have been steadily discredited it seems the CDC has decided to remove the page and it seems someone has been deleting the archived versions of the page from the web archive too].
Journal references:
1. Chess, S. Autism in children with congenital rubella. J Autism Child Schizophr. 1, 33-47 (1971).
2. Chess S. Follow-up report on autism in congenital rubella. J Autism Child Schizophr. 1977;7:69 –81
3. Ziring PR. Congenital rubella: the teenage years. Pediatr Ann. 1997;6: 762–770


Walker et al, 2013 New Publication showing autism &  gut issues, appears to be a solid study.